A single point mutation is the cause of the Greek form of hereditary persistence of fetal hemoglobin

By: Berry, MContributor(s): Dillon, N | Grosveld, FMaterial type: Text

TextReference number:a9352 In: Nature 358 (6386), 499-502.

Reviews from LibraryThing.com:

Tags from this library: No tags from this library for this title. Log in to add tags.

Holdings
Item type	Current library	Collection	Class number	Copy number	Status	Date due	Barcode	Item reservations
Magazine	Staff publications New Materials Shelf	Non-fiction	XX(9352.1) (Browse shelf(Opens below))	1	Available		9352-1001

Total reservations: 0

Previous	No cover image available	No cover image available	No cover image available	No cover image available	No cover image available	No cover image available	No cover image available	Next
Previous	XX(9349.1) Relative expression of surface IgM, IgD and the Ig-associating a(mb-1) and ß(B-29) polypeptide chains	XX(9350.1) Do antigenic drift residues in influenza hemagglutinins of the H3 subtype qualify as contact sites for MHC class II interaction?	XX(9351.1) The right end of the unique region of the genome of human herpesvirus-6 U1102 contains A candidate immediate early gene enhancer and a homolog of the human cytomegalovirus US22-gene family	XX(9352.1) A single point mutation is the cause of the Greek form of hereditary persistence of fetal hemoglobin	XX(9353.1) Proton nuclear magnetic resonance studies of the binding of sialosides to intact influenza virus	XX(9354.1) Thyroid hormone receptors and their role in development	XX(9355.1) Origins of the parasitophorous vacuole membrane of the malaria parasite Plasmodium faciparum, in human red blood cells	Next

There are no comments on this title.

to post a comment.

PTFS Europe